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The Netter Collection of Medical Illustrations: Musculoskeletal System, Volume 6, Part III - Biology and Systemic Diseases

The Netter Collection of Medical Illustrations: Musculoskeletal System, Volume 6, Part III - Biology and Systemic Diseases (Hardcover, 2 ed)

Frank H. Netter, Joseph P. Iannotti (엮은이)
  |  
Elsevier Science Health Science div
2013-03-09
  |  
172,230원

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The Netter Collection of Medical Illustrations: Musculoskeletal System, Volume 6, Part III - Biology and Systemic Diseases

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· 제목 : The Netter Collection of Medical Illustrations: Musculoskeletal System, Volume 6, Part III - Biology and Systemic Diseases (Hardcover, 2 ed) 
· 분류 : 외국도서 > 의학 > 해부학
· ISBN : 9781416063797
· 쪽수 : 368쪽

목차

SECTION 1?EMBRYOLOGY

DEVELOPMENT OF MUSCULOSKELETAL SYSTEM

1-1 Amphioxus and Human Embryo at 16

Days, 2

1-2 Differentiation of Somites into Myotomes,

Sclerotomes, and Dermatomes, 3

1-3 Progressive Stages in Formation of

Vertebral Column, Dermatomes, and

Myotomes; Mesenchymal Precartilage

Primordia of Axial and Appendicular

Skeletons at 5 Weeks, 4

1-4 Fate of Body, Costal Process, and Neural

Arch Components of Vertebral Column,

With Sites and Time of Appearance of

Ossification Centers, 5

1-5 First and Second Cervical Vertebrae at

Birth; Development of Sternum, 6

1-6 Early Development of Skull, 7

1-7 Skeleton of Full-Term Newborn, 8

1-8 Changes in Position of Limbs Before Birth;

Precartilage Mesenchymal Cell

Concentrations of Appendicular Skeleton

at 6 Weeks, 9

1-9 Changes in Ventral Dermatome Pattern

During Limb Development, 10

1-10 Initial Bone Formation in Mesenchyme;

Early Stages of Flat Bone Formation, 11

1-11 Secondary Osteon (Haversian

System), 12

1-12 Growth and Ossification of

Long Bones, 13

1-13 Growth in Width of a Bone and Osteon

Remodeling, 14

1-14 Remodeling: Maintenance of Basic

Form and Proportions of Bone During

Growth, 15

1-15 Development of Three Types of Synovial

Joints, 16

1-16 Segmental Distribution of Myotomes in

Fetus of 6 Weeks; Developing Skeletal

Muscles at 8 Weeks, 17

1-17 Development of Skeletal Muscle

Fibers, 18

1-18 Cross Sections of Body at 6 to

7 Weeks, 19

1-19 Prenatal Development of Perineal

Musculature, 20

1-20 Origins and Innervations of Pharyngeal

Arch and Somite Myotome Muscles, 21

1-21 Branchiomeric and Adjacent Myotomic

Muscles at Birth, 22

SECTION 2?PHYSIOLOGY

2-1 Microscopic Appearance of Skeletal

Muscle Fibers, 25

2-2 Organization of Skeletal Muscle, 26

2-3 Intrinsic Blood and Nerve Supply of

Skeletal Muscle, 27

2-4 Composition and Structure of

Myofilaments, 28

2-5 Muscle Contraction and Relaxation, 29

2-6 Biochemical Mechanics of Muscle

Contraction, 30

2-7 Sarcoplasmic Reticulum and Initiation of

Muscle Contraction, 31

2-8 Initiation of Muscle Contraction by Electric

Impulse and Calcium Movement, 32

2-9 Motor Unit, 33

2-10 Structure of Neuromuscular Junction, 34

2-11 Physiology of Neuromuscular

Junction, 35

2-12 Pharmacology of Neuromuscular

Transmission, 36

2-13 Physiology of Muscle Contraction, 37

2-14 Energy Metabolism of Muscle, 38

2-15 Muscle Fiber Types, 39

2-16 Structure, Physiology, and

Pathophysiology of Growth Plate, 40-41

2-17 Structure and Blood Supply of Growth

Plate, 42

2-18 Peripheral Fibrocartilaginous Element of

Growth Plate, 43

2-19 Composition and Structure of

Cartilage, 44

2-20 Bone Cells and Bone Deposition, 45

2-21 Composition of Bone, 46

2-22 Structure of Cortical (Compact) Bone, 47

2-23 Structure of Trabecular Bone, 48

2-24 Formation and Composition of

Collagen, 49

2-25 Formation and Composition of

Proteoglycan, 50

2-26 Structure and Function of Synovial

Membrane, 51

2-27 Histology of Connective Tissue, 52

2-28 Dynamics of Bone Homeostasis, 53

2-29 Regulation of Calcium and Phosphate

Metabolism, 54

2-30 Effects of Bone Formation and Bone

Resorption on Skeletal Mass, 55

2-31 Four Mechanisms of Bone Mass

Regulation, 56

2-32 Normal Calcium and Phosphate

Metabolism, 57

2-33 Nutritional Calcium Deficiency, 59

2-34 Effects of Disuse and Stress (Weight

Bearing) on Bone Mass, 60

2-35 Musculoskeletal Effects of Weightlessness

(Space Flight), 61

2-36 Bone Architecture and Remodeling in

Relation to Stress, 62

2-37 Stress-Generated Electric Potentials in

Bone, 63

2-38 Bioelectric Potentials in Bone, 64

2-39 Age-Related Changes in Bone

Geometry, 65

2-40 Age-Related Changes in Bone Geometry

(Continued), 66

SECTION 3?METABOLIC DISEASES

3-1 Parathyroid Hormone, 68

3-2 Pathophysiology of Primary

Hyperparathyroidism, 69

3-3 Clinical Manifestations of Primary

Hyperparathyroidism, 70

3-4 Differential Diagnosis of Hypercalcemic

States, 71

3-5 Pathologic Physiology of

Hypoparathyroidism, 72

3-6 Clinical Manifestations of Chronic

Hypoparathyroidism, 74

3-7 Clinical Manifestations of

Hypocalcemia, 75

3-8 Pseudohypoparathyroidism, 76

3-9 Mechanism of Parathyroid Hormone

Activity on End Organ, 77

3-10 Mechanism of Parathyroid Hormone

Activity on End Organ: Cyclic AMP

Response to PTH, 78

3-11 Clinical Guide to Parathyroid Hormone

Assay: Different Forms of PTH and Their

Detection by Whole (Bioactive) PTH and

I-PTH Immunometric Assays, 79

3-12 Clinical Guide to Parathyroid Hormone

Assay (Continued), 80

3-13 Childhood Rickets, 81

3-14 Adult Osteomalacia, 82

3-15 Nutritional Deficiency: Rickets and

Osteomalacia, 83

3-16 Vitamin D?Resistant Rickets and

Osteomalacia due to Proximal Renal

Tubular Defects (Hypophosphatemic

Rachitic Syndromes), 84

3-17 Vitamin D?Resistant Rickets and

Osteomalacia due to Proximal and Distal

Renal Tubular Defects, 85

3-18 Vitamin D?Dependent (Pseudodeficiency)

Rickets and Osteomalacia, 86

3-19 Vitamin D?Resistant Rickets and

Osteomalacia due to Renal Tubular

Acidosis, 87

3-20 Metabolic Aberrations of Renal

Osteodystrophy, 88

3-21 Rickets, Osteomalacia, and Renal

Osteodystrophy, 89

3-22 Bony Manifestations of Renal

Osteodystrophy, 90

3-23 Vascular and Soft Tissue Calcification in

Secondary Hyperparathyroidism of

Chronic Renal Disease, 91

3-24 Clinical Guide to Vitamin D

Measurement, 92

3-25 Hypophosphatasia, 93

3-26 Causes of Osteoporosis, 94

3-27 Involutional Osteoporosis, 95

3-28 Clinical Manifestations of

Osteoporosis, 96

3-29 Progressive Spinal Deformity in

Osteoporosis, 97

3-30 Radiology of Osteopenia, 98

3-31 Radiology of Osteopenia (Continued), 99

3-32 Radiology of Osteopenia (Continued), 100

3-33 Transiliac Bone Biopsy, 101

3-34 Treatment of Complications of Spinal

Osteoporosis, 102

3-35 Treatment of Osteoporosis, 103

3-36 Treatment of Osteoporosis

(Continued), 104

3-37 Osteogenesis Imperfecta Type I, 106

3-38 Osteogenesis Imperfecta Type III, 107

3-39 Marfan Syndrome, 108

3-40 Marfan Syndrome (Continued), 109

3-41 Ehlers-Danlos Syndromes, 110

3-42 Ehlers-Danlos Syndromes (Continued), 111

3-43 Osteopetrosis (Albers-Schonberg

Disease), 112

3-44 Paget Disease of Bone, 113

3-45 Paget Disease of Bone (Continued), 114

3-46 Pathophysiology and Treatment of Paget

Disease of Bone, 115

3-47 Fibrodysplasia Ossificans Progressiva, 116

SECTION 4?CONGENITAL AND

DEVELOPMENTAL DISORDERS

DWARFISM

4-1 Achondroplasia?Clinical

Manifestations, 118

4-2 Achondroplasia?Clinical Manifestations

(Continued), 119

4-3 Achondroplasia?Clinical Manifestations of

Spine, 120

4-4 Achondroplasia?Diagnostic Testing, 121

4-5 Hypochondroplasia, 122

4-6 Diastrophic Dwarfism, 123

4-7 Pseudoachondroplasia, 124

4-8 Metaphyseal Chondrodysplasia, McKusick

Type, 125

4-9 Metaphyseal Chondrodysplasia, Schmid

Type, 126

4-10 Chondrodysplasia Punctata, 127

4-11 Chondroectodermal Dysplasia (Ellis-van

Creveld Syndrome), Grebe

Chondrodysplasia, and Acromesomelic

Dysplasia, 128

4-12 Multiple Epiphyseal Dysplasia, Fairbank

Type, 129

4-13 Pycnodysostosis (Pyknodysostosis), 130

4-14 Camptomelic (Campomelic)

Dysplasia, 131

4-15 Spondyloepiphyseal Dysplasia Tarda

and Spondyloepiphyseal Dysplasia

Congenita, 132

4-16 Spondylocostal Dysostosis and Dyggve-

Melchior-Clausen Dysplasia, 133

4-17 Kniest Dysplasia, 134

4-18 Mucopolysaccharidoses, 135

4-19 Principles of Treatment of Skeletal

Dysplasias, 136

NEUROFIBROMATOSIS

4-20 Diagnostic Criteria and Cutaneous Lesions

in Neurofibromatosis, 137

4-21 Cutaneous Lesions in

Neurofibromatosis, 138

4-22 Spinal Deformities in

Neurofibromatosis, 139

4-23 Bone Overgrowth and Erosion in

Neurofibromatosis, 140

OTHER

4-24 Arthrogryposis Multiplex Congenita, 141

4-25 Fibrodysplasia Ossificans Progressiva and

Progressive Diaphyseal Dysplasia, 142

4-26 Osteopetrosis and Osteopoikilosis, 143

4-27 Melorheostosis, 144

4-28 Congenital Elevation of Scapula, Absence

of Clavicle, and Pseudarthrosis of

Clavicle, 145

4-29 Madelung Deformity, 146

4-30 Congenital Bowing of the Tibia, 147

4-31 Congenital Pseudoarthrosis of the Tibia

and Dislocation of the Knee, 148

LEG-LENGTH DISCREPANCY

4-32 Clinical Manifestations, 149

4-33 Evaluation of Leg-Length Discrepancy, 150

4-34 Charts for Timing Growth Arrest and

Determining Amount of Limb Lengthening

to Achieve Limb-Length Equality at

Maturity, 151

4-35 Growth Arrest, 152

4-36 Ilizarov and De Bastiani Techniques for

Limb Lengthening, 153

CONGENITAL LIMB MALFORMATION

4-37 Growth Factors, 154

4-38 Foot Prehensility in Amelia, 155

4-39 Failure of Formation of Parts: Transverse

Arrest, 156

4-40 Failure of Formation of Parts: Transverse

Arrest (Continued), 157

4-41 Failure of Formation of Parts: Transverse

Arrest (Continued), 158

4-42 Failure of Formation of Parts: Transverse

Arrest (Continued), 159

4-43 Failure of Formation of Parts: Transverse

Arrest (Continued), 160

4-44 Failure of Formation of Parts: Transverse

Arrest (Continued), 161

4-45 Failure of Formation of Parts: Transverse

Arrest (Continued), 162

4-46 Failure of Formation of Parts: Longitudinal

Arrest, 163

4-47 Failure of Formation of Parts: Longitudinal

Arrest (Continued), 164

4-48 Failure of Formation of Parts: Longitudinal

Arrest (Continued), 165

4-49 Failure of Formation of Parts: Longitudinal

Arrest (Continued), 166

4-50 Duplication of Parts, Overgrowth, and

Congenital Constriction Band

Syndrome, 167

SECTION 5?RHEUMATIC DISEASES

RHEUMATIC DISEASES

5-1 Joint Pathology in Rheumatoid

Arthritis, 170

5-2 Early and Moderate Hand Involvement in

Rheumatoid Arthritis, 171

5-3 Advanced Hand Involvement in

Rheumatoid Arthritis, 172

5-4 Foot Involvement in Rheumatoid

Arthritis, 173

5-5 Knee, Shoulder, and Hip Joint Involvement

in Rheumatoid Arthritis, 174

5-6 Extra-articular Manifestations in

Rheumatoid Arthritis, 175

5-7 Extra-articular Manifestations in

Rheumatoid Arthritis (Continued), 176

5-8 Immunologic Features in Rheumatoid

Arthritis, 177

5-9 Variable Clinical Course of Adult

Rheumatoid Arthritis, 178

TREATMENT OF RHEUMATOID ARTHRITIS

5-10 Exercises for Upper Extremities, 179

5-11 Exercises for Shoulders and Lower

Extremities, 180

5-12 Surgical Management in Rheumatoid

Arthritis, 181

SYNOVIAL FLUID EXAMINATION

5-13 Techniques for Aspiration of

Joint Fluid, 182

5-14 Synovial Fluid Examination, 183

5-15 Synovial Fluid Examination

(Continued), 184

JUVENILE ARTHRITIS

5-16 Systemic Juvenile Arthritis, 185

5-17 Systemic Juvenile Arthritis

(Continued), 186

5-18 Hand Involvement in Juvenile

Arthritis, 187

5-19 Lower Limb Involvement in Juvenile

Arthritis, 188

5-20 Ocular Manifestations in Juvenile

Arthritis, 189

5-21 Sequelae of Juvenile Arthritis, 190

OSTEOARTHRITIS

5-22 Distribution of Joint Involvement in

Osteoarthritis, 191

5-23 Clinical Findings in Osteoarthritis, 192

5-24 Clinical Findings in Osteoarthritis

(Continued), 193

5-25 Hand Involvement in Osteoarthritis, 194

5-26 Hip Joint Involvement in

Osteoarthritis, 195

5-27 Degenerative Changes, 196

5-28 Spine Involvement in Osteoarthritis, 197

OTHER

5-29 Ankylosing Spondylitis, 198

5-30 Ankylosing Spondylitis (Continued), 199

5-31 Ankylosing Spondylitis (Continued)

Degenerative Changes in the Cervical

Vertebrae, 200

5-32 Psoriatic Arthritis, 201

5-33 Reactive Arthritis (formerly Reiter

Syndrome), 202

5-34 Infectious Arthritis, 203

5-35 Tuberculous Arthritis, 204

5-36 Hemophilic Arthritis, 205

5-37 Neuropathic Joint Disease, 206

5-38 Gouty Arthritis, 207

5-39 Tophaceous Gout, 208

5-40 Articular Chondrocalcinosis

(Pseudogout), 209

5-41 Nonarticular Rheumatism, 210

5-42 Clinical Manifestations of Polymyalgia

Rheumatica and Giant Cell Arteritis, 211

5-43 Imaging of Polymyalgia Rheumatica and

Giant Cell Arteritis, 212

5-44 Fibromyalgia, 213

5-45 Pathophysiology of Autoinflammatory

Syndromes, 214

5-46 Cutaneous Findings in Autoinflammatory

Syndromes, 215

5-47 Joint and Central Nervous System Findings

in Autoinflammatory Syndromes, 216

5-48 Vasculitis: Vessel Distribution, 217

5-49 Vasculitis: Clinical and Histologic Features

of Granulomatosis with Polyangitis

(Wegener), 218

5-50 Key Features of Primary Vasculitic

Diseases, 219

5-51 Renal Lesions in Systemic Lupus

Erythematosus, 220

5-52 Cutaneous Lupus Band Test, 221

5-53 Lupus Erythematosus of the Heart, 222

5-54 Antiphospholipid Syndrome, 223

5-55 Scleroderma?Clinical Manifestations, 225

5-56 Scleroderma?Clinical Findings, 226

5-57 Scleroderma?Radiographic Findings of

Acro-osteolysis and Calcinosis Cutis, 227

5-58 Polymyositis and Dermatomyositis, 228

5-59 Polymyositis and Dermatomyositis

(Continued), 229

5-60 Primary Angiitis of the Central Nervous

System, 230

5-61 Behcet Syndrome, 232

5-62 Behcet Syndrome (Continued), 233

SECTION 6?TUMORS OF

MUSCULOSKELETAL SYSTEM

6-1 Initial Evaluation and Staging of

Musculoskeletal Tumors, 236

6-2 Osteoid Osteoma, 238

6-3 Osteoblastoma, 239

6-4 Enchondroma, 240

6-5 Periosteal Chondroma, 241

6-6 Osteocartilaginous Exostosis

(Osteochondroma), 242

6-7 Chondroblastoma and Chondromyxoid

Fibroma, 243

6-8 Fibrous Dysplasia, 244

6-9 Nonossifying Fibroma and Desmoplastic

Fibroma, 245

6-10 Eosinophilic Granuloma, 246

6-11 Aneurysmal Bone Cyst, 247

6-12 Simple Bone Cyst, 248

6-13 Giant Cell Tumor of Bone, 249

6-14 Osteosarcoma, 250

6-15 Osteosarcoma (Continued), 251

6-16 Osteosarcoma (Continued), 252

6-17 Chondrosarcoma, 253

6-18 Fibrous Histiocytoma and Fibrosarcoma of

Bone, 254

6-19 Reticuloendothelial Tumors?Ewing

Sarcoma, 255

6-20 Reticuloendothelial Tumors?

Myeloma, 256

6-21 Adamantinoma, 257

6-22 Tumors Metastatic to Bone, 258

6-23 Desmoid, Fibromatosis, and

Hemangioma, 259

6-24 Lipoma, Neurofibroma, and Myositis

Ossificans, 260

6-25 Sarcomas of Soft Tissue, 261

6-26 Sarcomas of Soft Tissue (Continued), 262

6-27 Sarcomas of Soft Tissue (Continued), 263

6-28 Tumor Biopsy, 264

6-29 Surgical Margins, 265

6-30 Reconstruction after Partial Excision

or Curettage of Bone (Fracture

Prophylaxis), 266

6-31 Limb-Salvage Procedures for

Reconstruction, 267

6-32 Radiologic Findings in Limb-Salvage

Procedures, 268

6-33 Limb-Salvage Procedures, 269

SECTION 7?INJURY TO

MUSCULOSKELETAL SYSTEM

7-1 Closed Soft Tissue Injuries, 272

7-2 Open Soft Tissue Wounds, 273

7-3 Treatment of Open Soft Tissue

Wounds, 274

7-4 Pressure Ulcers, 275

7-5 Excision of Deep Pressure Ulcer, 276

7-6 Classification of Burns, 277

7-7 Causes and Clinical Types of Burns, 278

7-8 Escharotomy for Burns, 279

7-9 Prevention of Infection in Burn

Wounds, 280

7-10 Metabolic and Systemic Effects of

Burns, 281

7-11 Excision and Grafting for Burns, 282

7-12 Etiology of Compartment Syndrome, 283

7-13 Pathophysiology of Compartment and

Crush Syndromes, 284

7-14 Acute Anterior Compartment

Syndrome, 285

7-15 Measurement of Intracompartmental

Pressure, 286

7-16 Incisions for Compartment Syndrome of

Forearm and Hand, 287

7-17 Incisions for Compartment Syndrome of

Leg, 288

7-18 Healing of Incised, Sutured Skin

Wound, 289

7-19 Healing of Excised Skin

Wound, 290

7-20 Types of Joint Injury, 291

7-21 Classification of Fracture, 292

7-22 Types of Displacement, 293

7-23 Types of Fracture, 294

7-24 Healing of Fracture, 295

7-25 Primary Union, 296

7-26 Factors That Promote or Delay Bone

Healing, 297

SECTION 8?SOFT TISSUE INFECTIONS

8-1 Septic Joint, 300

8-2 Etiology and Prevalence of Hematogenous

Osteomyelitis, 301

8-3 Pathogenesis of Hematogenous

Osteomyelitis, 302

8-4 Clinical Manifestations of Hematogenous

Osteomyelitis, 303

8-5 Direct (Nonhematogenous) Causes of

Osteomyelitis, 304

8-6 Direct (Nonhematogenous) Causes of

Osteomyelitis (Continued), 305

8-7 Osteomyelitis after Open Fracture, 306

8-8 Recurrent Postoperative

Osteomyelitis, 307

8-9 Delayed Posttraumatic Osteomyelitis in

Diabetic Patient, 308

SECTION 9?COMPLICATIONS

OF FRACTURE

9-1 Neurovascular Injury, 310

9-2 Adult Respiratory Distress

Syndrome, 311

9-3 Infection, 312

9-4 Surgical Management of Open

Fractures, 313

9-5 Gas Gangrene, 314

9-6 Implant Failure, 315

9-7 Malunion of Fracture, 316

9-8 Growth Deformity, 317

9-9 Posttraumatic Osteoarthritis, 318

9-10 Osteonecrosis, 319

9-11 Joint Stiffness, 320

9-12 Complex Regional Pain Syndrome, 321

9-13 Nonunion of Fracture, 322

9-14 Surgical Management of Nonunion, 323

9-15 Electric Stimulation of Bone Growth, 324

9-16 Noninvasive Coupling Methods of Electric

Stimulation of Bone, 325

저자소개

Frank H. Netter (지은이)    정보 더보기
Frank H. Netter는 1906년 뉴욕에서 태어나, Art Students League 과 National Academy of Design에서 미술을 전공하고 New York University에서 의학을 공부해 1931년 의학박사 학위를 받았다. 의학 공부를 할 때 교수와 다른 의사들이 Netter의 해부도에 관심을 가졌으며, Netter에게 비용을 지불하고 논문이나 교과서 그림을 요청하기도 했다. 그는 1933년 수술 병원을 설립한 후 부업으로 그림을 계속 그렸지만, 이후 그림에 몰두하기 위해 병원을 그만두었다. 제2차 세계대전 기간 동안 미군에서 복역한 후, Netter는 CIBA Pharmaceutical Company(현 Norvatis 제약회사)와 협력하기 시작했다. 45년간 계속된 협력으로 뛰어난 의학해부도 총서가 발간되었으며, 전 세계적으로 사용되고 있다. Icon Learning Systems에서 2000년 7월, the Netter Collection을 준비하고 Netter의 원본 도해를 지속적으로 업데이트하였 으며 Netter식 교습을 받은 도해가들의 새로운 그림을 추가하였 다. 2005년, Elsevier가 The Netter Collection과 함께 Icon Learning Systems 사의 모든 출판물을 인도했다. 현재 Elsevier의 이름으로 Netter의 도해를 사용한 책은 50권이 넘는다. Netter의 그림은 의학 강의 내에 사용할 수 있는 가장 정확하고 섬세한 도해이다. 13권 세트인 <The Netter Collection of Medical Illustrations>는 Netter가 그린 2만여 장의 도해가 실려 있고, 지금까지 의학 분야 세계 최고의 도해서로 명성을 알리고 있다. <The Netter Atlas of Human Anatomy>는 1989년에 처음 출간 하여 ‘Netter Collection’의 해부학 도해를 사용하고 있다. 16개국 언어로 번역된 <Atlas of human anatomy>는 전세계 의학 및 보건 학생이 가장 많이 보는 해부학 도해서이다. ‘The Netter illustrations’는 미적 요소는 물론 지식적 요소도 그 품질이 높다. Netter는 1949년 “도해서의 목표는 주제 분류이다. 그림이 얼마나 아름답고 얼마나 섬세하든, 의학적인 요소가 확실하지 않으면 의학적으로 아무런 쓸모가 없을 것이다”라는 말을 남겼다. Netter의 계획, 구상, 관점, 접근은 그의 도해에 그대로 반영되었으며 그 가치를 더욱 올려주었다. 의사이자 도해가인 Frank H. Netter, MD는 1991년 별세했다.
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